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Therapeutic Opportunities Presented by Modulation of Cellular Senescence
Richard G.A. Faragher
,
Neda Heidari
, Elizabeth L. Ostler
School of Applied Sciences
Research output
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Chapter in Book/Conference proceeding with ISSN or ISBN
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Chapter
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peer-review
Overview
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Keyphrases
Ablation
25%
Age-related Illnesses
50%
Aging Mechanism
25%
Antagonistic Pleiotropy
25%
Bioartificial Organs
25%
Bioinformatics Approach
25%
Cell Senescence
25%
Cellular Aging
25%
Cellular Senescence
100%
Chronic Disability
25%
Chronic Disease
25%
Evolution of Aging
25%
Growth Arrest
25%
Later Life
25%
P38 MAP Kinase
25%
Permanent State
25%
Physiological Processes
25%
Polyphenols
25%
Premature Aging
25%
Process Level
25%
Senescence
75%
Senescent Cells
100%
Text Mining
25%
Therapeutic Potential
100%
Transgenic Mice
25%
Translational Medicine
25%
Tumor Suppression
25%
Pharmacology, Toxicology and Pharmaceutical Science
Cancer Inhibition
50%
Chronic Disease
50%
Diseases
100%
Growth Disorder
50%
Mitogen Activated Protein Kinase P38
50%
Premature Aging
50%
Transgenic Mouse
50%
Neuroscience
Cellular Senescence
100%
Growth Disorder
33%
P38 Mitogen-Activated Protein Kinases
33%
Biochemistry, Genetics and Molecular Biology
Antagonistic Pleiotropy
50%