Increased linear bone growth by GH in the absence of SOCS2 is independent of IGF-1

R Dobie; SF Ahmed; KA Staines; C Pass; S Jasim; VE MacRae; C Farquharson

doi:10.1002/jcp.25006

Increased linear bone growth by GH in the absence of SOCS2 is independent of IGF-1

R Dobie, SF Ahmed, KA Staines, C Pass, S Jasim, VE MacRae, C Farquharson

School of Pharmacy and Biomolecular Sci

Research output: Contribution to journal › Article › peer-review

Original language	English
Pages (from-to)	2796-2806
Journal	Journal of Cellular Physiology
Volume	230
Issue number	11
DOIs	https://doi.org/10.1002/jcp.25006
Publication status	Published - 1 Apr 2015

Access to Document

10.1002/jcp.25006Licence: CC BY

Dobie_et_al-2015-Journal_of_Cellular_PhysiologyFinal published version, 1.53 MBLicence: CC BY

http://europepmc.org/abstract/med/25833299

Katherine Staines
- K.Staines@brighton.ac.uk
- School of Pharmacy and Biomolecular Sci - Senior Lecturer
- Centre for Stress and Age-Related Disease
Person: Academic

Cite this

@article{5d5465871e6547ffa2799887cf2693ee,

title = "Increased linear bone growth by GH in the absence of SOCS2 is independent of IGF-1",

author = "R Dobie and SF Ahmed and KA Staines and C Pass and S Jasim and VE MacRae and C Farquharson",

year = "2015",

month = apr,

day = "1",

doi = "10.1002/jcp.25006",

language = "English",

volume = "230",

pages = "2796--2806",

journal = "Journal of Cellular Physiology",

issn = "0021-9541",

number = "11",

}

TY - JOUR

T1 - Increased linear bone growth by GH in the absence of SOCS2 is independent of IGF-1

AU - Dobie, R

AU - Ahmed, SF

AU - Staines, KA

AU - Pass, C

AU - Jasim, S

AU - MacRae, VE

AU - Farquharson, C

PY - 2015/4/1

Y1 - 2015/4/1

UR - http://europepmc.org/abstract/med/25833299

U2 - 10.1002/jcp.25006

DO - 10.1002/jcp.25006

M3 - Article

C2 - 25833299

VL - 230

SP - 2796

EP - 2806

JO - Journal of Cellular Physiology

JF - Journal of Cellular Physiology

SN - 0021-9541

IS - 11

ER -

Increased linear bone growth by GH in the absence of SOCS2 is independent of IGF-1

Access to Document

Katherine Staines

Cite this